Cindy Voisine and Disease - Free people check

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Wellfleet library puts women scientists at center stage

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Mar 11, — ... and Cindy Voisine, who will discuss her work researching the causes of age-related neurodegenerative diseases such as Alzheimer's, ... › story › › well...

Modification Of Mutant Huntingtin Protein Increases Its Clearance...

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A new study has identified a potential strategy for removing the abnormal protein that causes Huntington's disease from brain cells, which could slow the...

Cindy VOISINE | Assistant Professor | Northeastern Illinois ...

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Cindy Voisine Protein homeostasis, or proteostasis, is fundamental to cellular and organismal health. Proteostasis collapse is linked to diverse diseases, including neurodegeneration and cancers.

Trinucleotide Repeat Protocols - Page Google Books Result

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10 Caenorhabditis elegans as a Model System for Triplet Repeat Diseases Cindy Voisine and Anne C. Hart Summary Common features underlie the generation and ...

Heat Shock Protein 90 in Human Diseases and Disorders - Google Books

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The book Heat Shock Protein 90 in Human Diseases and Disorders provides the most comprehensive review on contemporary knowledge on the role of HSP90. Using an...

Journal of Clinical Tuberculosis and Other Mycobacterial Diseases ...

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Mário F. Neto, Quan H. Nguyen, Joseph Marsili, Sally M. McFall, Cindy Voisine. Page 8: Download PDF. select article Why has MDR-TB prevalence increased in ...

Caenorhabditis elegans as a Model System for Triplet Repeat Diseases...

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Common features underlie the generation and function of neurons in multicellular animals. It is likely that conserved pathways and genes also are involved in...

Neurodegeneration - an overview | ScienceDirect Topics

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Cindy Voisine, ... Richard I. Morimoto, in Molecular Neurology, B. Parkinson Disease Models. Neurodegeneration has been associated with abnormal protein folding and toxicity for many disorders besides polyQ diseases. Aggregation of α-synuclein is associated with Parkinson disease (Polymeropoulos et al., 1997) (see Table 5.1).